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Inflammatory stimuli induce inhibitory S-nitrosylation of the deacetylase SIRT1 to increase acetylation and activation of p53 and p65

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Defining conformational ensembles of intrinsically disordered and partially folded proteins directly from chemical shifts

Animal studies have demonstrated that Fmr1 knockout mice have fewer GABAergic synapses, reduced expression levels of GABAA receptors, and downregulation of GAD65 and GAD67 transcription

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